Atrial Contractions

Christian asked:


My mother has been diagnosed with Premature Atrial Contractions, and ive been real worried. Can someone die from this?

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Sam asked:


a) to cause atrial contraction
b) to act as a pacemaker and initiate the heartbeat
c) to prevent the valves from inverting during the heartbeat
d) to conduct impulses from the AV node to the ventricles

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Juliet Cohen asked:


Ebstein’s anomaly is a heart defect in which the tricuspid valve is abnormally formed. In Ebstein’s anomaly, two leaflets of the tricuspid valve are displaced downward into the pumping chamber and the third leaflet is elongated and may be adherent to the wall of the chamber. In addition, many patients with Ebstein’s anomaly have an accessory conduction pathway in the heart leading to episodes of abnormal fast heart rate. These abnormalities cause the tricuspid valve to leak blood backwards into the right atrium when the right ventricle contracts and as a result, the right atrium becomes enlarged and if severe enough, congestive heart failure can result. There appears to be an increased risk of this abnormality in infants of women taking lithium during the first trimester of pregnancy, and in those with wolff-parkinson-white syndrome.

When the Ebstein anomaly is defined took the tricuspid valve the congenital displacement toward the right ventricle apex, it frequently with other abnormalities relates in together. Ebstein’s anomaly may also lead to enlargement of the heart. In addition, about half the people with Ebstein’s anomaly have a hole in their heart, and one in four has episodes of fast heartbeats. If no signs or symptoms are present, careful monitoring of your heart may be all that’s necessary. In Ebstein’s anomaly, the tricuspid valve is displaced downward into the right ventricle so that a portion of the right ventricle becomes part of the right atrium. As a result, the right atrium is larger than usual and the functional right ventricle is abnormal. Other tachyarrhythmias, such as atrial fibrillation and atrial flutter, occur with increased frequency as the right atrium becomes dilated. In some cases, a very fast heart rhythm may cause fainting spells.

Ebstein anomaly possibility realm from extremely temperate, by the small symptom, to is extremely severe. Many patients with milder forms of Ebstein anomaly do not have symptoms to be certain because of the presence of a heart noise. People with Ebstein’s anomaly may have a rapid heart rhythm called supraventricular tachycardia. An episode of supraventricular tachycardia may cause palpitations. Sometimes this is associated with fainting, dizziness, lightheadedness or chest discomfort. If the valve abnormality is especially severe, you may have decreased stamina, fatigue, cyanosis, and sometimes fluid retention. These problems usually develop because the valve has become leakier. Some babies and children have bluish discoloration to their lips and nail beds, due to the flow of blood from the right atrium to the left atrium. Severely affected babies are often critically ill at birth, with low oxygen saturations and heart failure requiring intensive care.

Some patients with abnormal heart rhythms can test the electrophysiologic to require potentially their problems of heart rhythm improve to identify and treat. The mild shortages can require no specific treatment, only disease prevention for bacterial endocarditis. Medical treatment is used to help children with congestive heart failure or abnormal heart rhythms. Ebstein’s anomaly is mild in most adults who have it, so they don’t need surgery. However sometimes the tricuspid valve leaks severely enough to result in heart failure or cyanosis. Then surgery may be required. Several different operations have been used in patients with Ebstein’s anomaly. The most common involves a repair of the tricuspid valve. People with Ebstein’s anomaly should receive continued care from a cardiologist with expertise in congenital heart defects. You may need antibiotics before certain dental or surgical procedures to prevent endocarditis.



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Aaron H asked:


im just getting my wisdom teeth pulled

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Aaron asked:


Anyone experience the following symptoms after eating meals:
Racing heart (palpitations), PVCs (premature ventricular contractions), bloated feeling in mid-section (above stomach), nausea/queasiness, diarrhea, fatigue, anxiety?

Last month I felt this way after eating, and all of a sudden I got dry heaves and my heart suddenly “jumped” into an arrhythmia while I was trying to throwup. Doctors said it was a very “chaotic” episode of atrial fibrillation. My heart finally converted to a normal sinus rhythm after enduring a long, 18-hour ordeal. Now after everytime I eat anything, or drink anything, I almost start to feel this way again and it depresses me. I eat very little throughout the day because of it. It doesn’t matter what I eat or drink either. From drinking alot of water, to eating an apple, to eating fast food, to whatever, I still get this way. Don’t ’spose anyone else has a similar experience?
Very interesting, patsy. I also find that if I sit for extended periods of time, my stomach feels unusually compressed and tight, as if my stomach is pushed “upwards” into my mid-section, also feels like I have alot of belching to do, but can’t, and my heart tends to skip and race during this, and of course if I eat, it makes it worse..I can definitely see the connection of a possible hiatal hernia causing my A-Fib episodes.Thanks for your insight

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book worm asked:


a) atrial depolarization
b) atrial repolarization
c) ventricular depolarization
d)ventricular repolarization
e) ventricular contraction

i think its d) ventricular repolarization but I’m not sure. If I’m wrong, can you correct me and tell me why? THANKS!

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Kaka asked:


… Junctional tachycardia, Premature junctional contraction, Junctional escape rhythm and Accelerated junctional rhythm ?

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Bill Chandler asked:


A mimicker is a symptom or disorder that fools its victim, as well as medical and psych professionals, into believing he/she is suffering from something other than the true cause of the problem. And the likeness is so striking that the mimicked issue gets all the attention, allowing the real culprit to remain a mystery. Obviously, mimickers can cause all sorts of confusion, as well as unnecessary pain and anguish. One of the major mimickers of panic attacks and panic disorder is mitral valve prolapse (MVP) and mitral valve prolapse syndrome (MVPS).

As always, let’s start with definitions. If the heart’s mitral valve is functioning properly, blood flows from the left atrium to the left ventricle in a very tidy manner. And that’s because blood is prevented from going back into the left atrium by a tight seal formed as the mitral valve closes. But if one has MVP the flaps of the mitral valve allow small amounts of blood leakage because the valve flaps don’t close evenly; one, or both, collapsing backward.

The physical manifestations of MVP may include heart palpitations, atrial fibrillation (the atria, the heart’s two small upper chambers, quiver instead of maintaining a normal rhythm, resulting in blood pooling and clotting because healthy pumping is interrupted - a stroke may ensue if a bit of blood clot breaks away and lodges in a brain artery), fainting, chest pain, and shortness of breath. MVP, a common and generally benign condition that presents in women three times more than men, is a genetic disorder typically confirmed by an echocardiogram. The only real potential for trouble is the very remote possibility of contracting an infection called endocarditis (an infection of the inner lining of the heart, the endocardium, and the possibility of blood clotting).

Now, here’s where things start to get panic-interesting. Just as a percentage of panic attack sufferers become panic disorder sufferers, so it is with MVP. Some 40% of patients with MVP also have something known as dysautonomia, an imbalance of the autonomic nervous system (ANS), which would indicate neurotransmitter and hormonal confusion as well. Well, the ANS has two components, the sympathetic and parasympathetic nervous systems. The sympathetic nervous system has the responsibility of ramping-up our biochemistry as our fight/flight response is switched on; and the parasympathetic nervous system returns our biochemistry to a state of normalcy as the threat is gone.

Indeed, an incredible number of bodily functions are directed by the ANS and when this system is out of balance the physiological results can be, as you may already know, panic attacks, anxiety, fatigue, migraine headache, irritable bowel syndrome, and many more little goodies. Well, when the situation becomes this complex one is said to suffer from mitral valve prolapse syndrome (MVPS). And it’s estimated that 40-60% of MVPS sufferers will experience panic attacks.

Are you saying to yourself, “Hmmm?” If you have ever suspected you suffer from mitral valve prolapse, or if members of your family have been diagnosed, please get to a doctor and have an evaluation. And even if you don’t have a family history, get tested anyway. A diagnosis of mitral valve prolapse or MVPS, and appropriate treatment, could dispose of your panic attacks and save you a whole lot of aggravation.

Indeed, perhaps you suffer from mitral valve prolapse or mitral valve prolapse syndrome, not panic. Women - are you paying attention?



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Cooler asked:


If so, show me a detail medical reason for this phenonmenon. Why doesn’t the Atria have a valve to prevent back flow if the atria do contract. Where does the blood go? What is the mechanism of atrial contraction ?> Please give a most detailed as answer as possible. I need to proove this to a fellow classmate who refuses to believe the atria contract, he says its not possible because if the atria contract blood will flow back as there is no valve that sepeates the atra from the vena cavas.

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Ben Escomm asked:


In this article we are focusing on the heart itself and what is actually taking place in the chambers during afib. But first lets go to a normal heart for a second.

In a normal heart we have a “lub-dubb” sequence. If you’re not familiar with what I’m talking about, you can find a full explanation on my website but here’s a quick description so you get the idea:

The top part of your heart (atria) squeezes and makes a “lub” sound, then the bottom part of your heart (ventricles) squeezes to give the “dubb” sound. “Lub-dubb” is the sequence that comes from one beat of the heart.

Now that’s outta the way, on to the rest of the article:

The lub is created by a electrical pulse in the right atrium (right upper chamber). See, the heart has its own natural pacemaker called the SA node-its basically a part of that chamber which automatically fires an electrical pulse that causes the heart to contract or squeeze-first in the atria (lub) and then in the ventricles (dub). The SA node controls the rate at which the heart beats in a normal heart.

In a heart with atrial fibrillation the electrical pulse that is fired from the SA Node isn’t the only pulse fired. There are other sites in the right atrium called foci (plural for focus) that are firing pulses as well. These other sites are firing faster and at the same time as your natural pacemaker causing multiple contractions of your upper chambers, which is why, during afib, the atria looks like jello quivering (lublublublllubuubllublub) as opposed to a single unit making a single squeeze (lub).

Its these multiple pulses from the right atrium which cause the ventricles (and therefore your heart rate) to be irregular…and, as you recall it’s the AV node which helps slow the rate of contraction from the atria to the ventricles.

Need more explanation of the concepts I presented in this article? Click here to get a better idea with additional descriptions, pictures, and animation.

www.understandingatrialfibrillation.moogo.com



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